A team of Bangladesh Army has detained seven members of Detective Branch of police, accused of collecting ransom after kidnapping a businessman in Teknaf upazila of Cox’s Bazar.The army detained the detectives with Tk 1.7 million cash on Wednesday morning.Major Nazim Ahmed of Sabrang Relief Centre at Teknaf led the army team that detained the DB men.Major Nazim said the DB team abducted businessman Abdul Gafur from Cox’s Bazar on Tuesday and demanded Tk 5 million as ransom.His family members in the end agreed to pay Tk 1.7 million for his release. After receiving the money, the detectives freed him in Marine Drive area of Cox’s Bazar.Informed by the victim’s family, the army team halted a DB police’s vehicle at an army check-post in Lambari area of the Cox’s Bazar Marine Drive.Maj Nazim said a sub-inspector managed to flee the scene, but others were caught red-handed.The detectives were taken to Sabrang army camp in the early hours of Wednesday.He said, later, the Cox’s Bazar district police superintendent and the additional police superintendent took the detainees from the army camp.The police supers said the authorities would take departmental action against the accused, he added.Officials at the police headquarters in Dhaka said the detained members of the DB are now in police custody.Afruzul Haque, the additional superintendent of police in Cox’s Bazar, could not be reached after repeated attempts for a comment on the issue.Later in the day, the authorities suspended the seven members of DB policeAdditional superintendent of Cox’s Bazar police Afruzul Haq Tutul told Prothom Alo that departmental action will be taken against the suspended detectives.
Firefighters work at the scene of a fire that broke out at a chemical warehouse in Dhaka, Bangladesh 21 February 2019. Photo: ReutersDozens of people were killed in Old Dhaka on Thursday in a blaze that ripped through apartment buildings where chemicals were stored.Fires and building collapses are common in the country — particularly in the multi-billion dollar garment industry — where building regulations are lax and volatile chemicals are often improperly kept.Here are some of the worst incidents in Bangladesh.Chemical fireThis building is burnt in Nimtali fire on 3 June in 2010. Photo: Prothom AloBangladesh’s deadliest industrial blaze was in Nimtali area in Dhaka in June 2010, when 117 people died in a fire that ripped through one of Dhaka’s most densely populated areas.The fire — fuelled by a chemical warehouse — destroyed several multi-storey apartment buildings and gutted a string of small shops.Industrial blazeFire broke out at Tazreen Fashion factory on 24 November 2012. File PhotoA fire swept through a nine-story garment factory near Dhaka in November 2012 killing 111 workers. An investigation later found it was caused by sabotage and managers at the plant prevented victims from escaping.”I broke open an exhaust fan in the second floor and jumped to the roof of a shed next to the factory,” said one survivor. “I broke my hand but survived somehow.”Packaging factory fireThirty-one people were killed when a fire triggered by a boiler explosion tore through a packaging factory north of the capital in September 2016.The blaze is thought to have spread quickly because of chemicals stored on the ground floor.Faulty boilerAn industrial boiler exploded at a factory killing 13 people and causing part of the six-storey building to collapse in July 2017.Most of the plant’s 5,000 workers were off for the Eid holidays when the boiler exploded during maintenance work.
Scanning electron microscope image of V. cholerae. Credit: public domain Journal information: Science Advances © 2016 Phys.org PausePlay% buffered00:0000:00UnmuteMuteDisable captionsEnable captionsSettingsCaptionsDisabledQuality0SpeedNormalCaptionsGo back to previous menuQualityGo back to previous menuSpeedGo back to previous menu0.5×0.75×Normal1.25×1.5×1.75×2×Exit fullscreenEnter fullscreen Citation: Study reveals how cholera’s two chromosomes communicate to coordinate replication (2016, April 25) retrieved 18 August 2019 from https://phys.org/news/2016-04-reveals-cholera-chromosomes-replication.html Explore further More information: M.-E. Val et al. A checkpoint control orchestrates the replication of the two chromosomes of Vibrio cholerae, Science Advances (2016). DOI: 10.1126/sciadv.1501914AbstractBacteria with multiple chromosomes represent up to 10% of all bacterial species. Unlike eukaryotes, these bacteria use chromosome-specific initiators for their replication. In all cases investigated, the machineries for secondary chromosome replication initiation are of plasmid origin. One of the important differences between plasmids and chromosomes is that the latter replicate during a defined period of the cell cycle, ensuring a single round of replication per cell. Vibrio cholerae carries two circular chromosomes, Chr1 and Chr2, which are replicated in a well-orchestrated manner with the cell cycle and coordinated in such a way that replication termination occurs at the same time. However, the mechanism coordinating this synchrony remains speculative. We investigated this mechanism and revealed that initiation of Chr2 replication is triggered by the replication of a 150-bp locus positioned on Chr1, called crtS. This crtS replication–mediated Chr2 replication initiation mechanism explains how the two chromosomes communicate to coordinate their replication. Our study reveals a new checkpoint control mechanism in bacteria, and highlights possible functional interactions mediated by contacts between two chromosomes, an unprecedented observation in bacteria. V. cholera has been studied extensively for some time as part of an ongoing process to either eliminate it as a threat to people, or to treat infections. As part of that effort, scientists have found that each bacterium has two chromosomes, both of which need to split prior to the cell splitting as part of its life cycle. Prior research has shown that the chromosomes split at the same time, which has suggested a means of communications between the two. In this new effort, the researchers have identified that means of communication and the parts of the chromosomes that are involved in the process.The two chromosomes in each cell are called chr1 and chr2. Chr1 is a lot longer than chr2 and is the chromosome that initiates the entire division process, the team found, when a DnaA molecule binds to a part of the chromosome called ori2 leading to the replication process in chr1, along a circular path. When that path crosses a point the researchers have called crtS, a molecule called RctB is activated which makes its way to chr2 causing it to begins its replication process. The two processes then finish at very nearly the same time—just prior to the complete cell dividing—due to the physical proximity of the two chromosomes and the location of the crtS on chr1.The team then deleted the crtS region in some of the bacteria and found that doing so caused problems with growth and DNA damage. They also allowed one group of the mutant bacteria to multiply over 200 generations to learn more about how the bacteria might compensate for the loss of crtS and discovered that that the mutations led only to mutations in RctB, which the team claims, shows that activation by RctB is a crucial part of the process. (Phys.org)—A team of researchers from France and Denmark, working at Institut Pasteur, in Paris has found the mechanism that is involved that allows dual chromosomes in Vibrio cholerae cells to split at the same time as part of replication. In their paper published in the journal Science Advances, the team describes how they closely studied cell replication in V. cholerae, the bacteria that cause cholera, and then verified their understanding of how chromosome division was occurring by disabling different parts involved in the process. Researchers discover novel role of TBK1 protein in cell division Play An animation presenting the model for replication checkpoint control in Vibrio cholerae. Credit: Val Marie-Eve This document is subject to copyright. Apart from any fair dealing for the purpose of private study or research, no part may be reproduced without the written permission. The content is provided for information purposes only.